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  1. A study in the journal, Human & Experimental Toxicology (Oct. 2012) examined FDA’s Vaccine Adverse Event Reporting System (VAERS) database and found an increase in reports of spontaneous abortion and stillbirths following pregnant woman’s vaccination against the flu and H1N1.

  2. A report in the journal Toxicological & Environmental Chemistry (2012) found that the datademonstrated that Hg [mercury] exposures, particularly during the first trimester of pregnancy, at well-established dose/weight ratios produced severe damage to humans including death.”

3. A third report in the journal Archives of Pediatric & Adolescent Medicine (a JAMA Network journal) reports that prenatal exposure to mercury and fish is linked to behavior associated with Attention Deficit Hypertension Disorder.

The evidence is clear that mercury is toxic for children’s neurological development.

Although the government-industrial vaccine complex never acknowledged the harm produced by mercury (Thimerisol) toxicity in children’s vaccines, it was removed from most childhood vaccines.

It should not come as a surprise that mercury is harmful to developing human embryos–whether the mercury exposure is from fish, water, or vaccine adjuvants.

What then are officials at the Centers for Disease Control thinking when they recommend the flu vaccine–that contains mercury–for pregnant women?

Given that the flu is hardly a life-threatening illness, and given that the evidence of efficacy for the flu vaccine has been found to be negligible–at best–as determined by the Cochrane review, we question the morally irresponsible, medically unsupportable CDC recommendation urging pregnant women to get vaccinated with a vaccine containing mercury.

CDC officials who formulated the flu vaccine policy urging pregnant women to be vaccinated with a vaccine containing mercury–in complete disregard of the scientific evidence showing profound harm for the unborn–should be fired and the recommendation rescinded.


Vera Sharav

 Centers for Disease Control recommendation for pregnant women: cdc_recommendation-_flu_-_pregnant_women

Pregnant Women & Influenza (Flu)

Flu is more likely to cause severe illness in pregnant women than in women who are not pregnant. Changes in the immune system, heart, and lungs during pregnancy make pregnant women more prone to severe illness from flu as well as hospitalizations and even death. Pregnant woman with flu also have a greater chance for serious problems for their unborn baby, including premature labor and delivery.

Flu shots will protect pregnant women, their unborn babies and even protect the baby after birth.

The Flu Shot is the Best Protection Against Flu

Getting a flu shot is the first and most important step in protecting against flu. The flu shot given during pregnancy has been shown to protect both the mother and her baby (up to 6 months old) from flu. (The nasal spray vaccine should not be given to women who are pregnant.) Learn more about the flu vaccine.

The Flu Shot is Safe for Pregnant Women

Flu shots are a safe way to protect the mother and her unborn child from serious illness and complications of flu. The flu shot has been given to millions of pregnant women over many years. Flu shots have not been shown to cause harm to pregnant women or their babies. It is very important for pregnant women to get the flu shot.

1. GS Goldman.
Comparison of VAERS fetal-loss reports during three consecutive influenza seasons,
Human & Experimental Toxicolog
y, October, 2012

Was there a synergistic fetal toxicity associated with the two-vaccine 2009/2010 season?

Gary S Goldman, Independent Computer Scientist, P.O. Box 847, Pearblossom, CA 93553, USA Email:


The aim of this study was to compare the number of inactivated-influenza vaccine–related spontaneous abortion and stillbirth (SB) reports in the Vaccine Adverse Event Reporting System (VAERS) database during three consecutive flu seasons beginning 2008/2009 and assess the relative fetal death reports associated with the two-vaccine 2009/2010 season. The VAERS database was searched for reports of fetal demise following administration of the influenza vaccine/vaccines to pregnant women. Utilization of an independent surveillance survey and VAERS, two-source capture–recapture analysis estimated the reporting completeness in the 2009/2010 flu season. Capture–recapture demonstrated that the VAERS database captured about 13.2% of the total 1321 (95% confidence interval (CI): 815–2795) estimated reports, yielding an ascertainment-corrected rate of 590 fetal-loss reports per million pregnant women vaccinated (or 1 per 1695). The unadjusted fetal-loss report rates for the three consecutive influenza seasons beginning 2008/2009 were 6.8 (95% CI: 0.1–13.1), 77.8 (95% CI: 66.3–89.4), and 12.6 (95% CI: 7.2–18.0) cases per million pregnant women vaccinated, respectively. The observed reporting bias was too low to explain the magnitude increase in fetal-demise reporting rates in the VAERS database relative to the reported annual trends. Thus, a synergistic fetal toxicity likely resulted from the administration of both the pandemic (A-H1N1) and seasonal influenza vaccines during the 2009/2010 season.

<a href="">2. Ian A. Brown</a> & <a href="">David W. Austin</a><small><small>.
<big><big><big>Maternal transfer of mercury to the developing embryo/fetus: is there a safe level?</big></big></big></small></small><i><small>

<b><i>Toxicological & Environmental Chemistry</i></b>  <a href=""><span style="color: #000000;">Vol</span> <span style="color: #000000;">94</span> </a> 2012

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<p class="first last">Mercury (Hg) exposure is ubiquitous in modern society via vaccines, fish/crustacea, dental amalgam, food, water, and the atmosphere. This article examines Hg exposure in the context of primary exposure to pregnant women and secondary exposure experienced by their unborn babies. Babies <i>in utero</i> are particularly at risk of higher Hg exposure than adults (on a dose/weight basis through maternal Hg transfer via the placenta), and are more susceptible to adverse effects from mercury and its biologically active compounds. It is, therefore, critical that regulatory advisories around maximum safe Hg exposures account for pregnant women and secondary exposure that children <i>in utero</i> experience. This study focused on standardized embryonic and fetal Hg exposures via primary exposure to the pregnant mother of two common Hg sources (dietary fish and parenteral vaccines). <b>Data demonstrated that Hg exposures, particularly during the first trimester of pregnancy, at well-established dose/weight ratios </b><b>produced severe damage to humans including death. </b></p>

<p class="first last">In light of research suggestive of a mercuric risk factor for childhood conditions such as tic disorders, cerebral palsy, and <a href="">autism</a>, it is essential that Hg advisories account for secondary prenatal human exposures.</p>


  1. Sharon K. Sagiv, PhD, MPH; Sally W. Thurston, PhD; David C. Bellinger, PhD, MS; Chitra Amarasiriwardena, PhD; Susan A. Korrick, MD, MPH
    Prenatal Exposure to Mercury and Fish Consumption During Pregnancy and Attention-Deficit/Hyperactivity Disorder–Related Behavior in Children,
    Archives of Pediatrics & Adolescent Medicine [JAMA Network] October, 2012.


Objective  To investigate the association of prenatal mercury exposure and fish intake with attention-deficit/hyperactivity disorder (ADHD)–related behavior.

Methods  For a population-based prospective birth cohort recruited in New Bedford, Massachusetts (1993-1998), we analyzed data for children examined at age 8 years with peripartum maternal hair mercury measures (n = 421) or maternal report of fish consumption during pregnancy (n = 515). Inattentive and impulsive/hyperactive behaviors were assessed using a teacher rating scale and neuropsychological testing.

Results  The median maternal hair mercury level was 0.45 μg/g (range, 0.03-5.14 μg/g), and 52% of mothers consumed more than 2 fish servings weekly. In multivariable regression models, mercury exposure was associated with inattention and impulsivity/hyperactivity; some outcomes had an apparent threshold with associations at 1 μg/g or greater of mercury. For example, at 1 μg/g or greater, the adjusted risk ratios for mild/markedly atypical inattentive and impulsive/hyperactive behaviors were 1.4 (95% CI, 1.0-1.8) and 1.7 (95% CI, 1.2-2.4), respectively, for an interquartile range (0.5 μg/g) mercury increase; there was no confounding by fish consumption. For neuropsychological assessments, mercury and behavior associations were detected primarily for boys. There was a protective association for fish consumption (>2 servings per week) with ADHD-related behaviors, particularly impulsive/hyperactive behaviors (relative risk = 0.4; 95% CI, 0.2-0.6).

Conclusions  Low-level prenatal mercury exposure is associated with a greater risk of ADHD-related behaviors, and fish consumption during pregnancy is protective of these behaviors. These findings underscore the difficulties of balancing the benefits of fish intake with the detriments of low-level mercury exposure in developing dietary recommendations in pregnancy.



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